Laurie S Jeffers headshot

Laurie S Jeffers


Clinical Assistant Professor

1 212 998 5310

433 First Avenue
New York, NY 10010
United States

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Professional overview

Laurie S. Jeffers, DNP FNP-BC, is a clinical assistant professor at NYU Rory Meyers College of Nursing. With her extensive clinical expertise in the areas of adolescent health, perimenopause, menopause, and hormone replacement therapy, she has been a board-certified family nurse practitioner since 1998 and a specialist in women's health for the last 20 years. She created the first women's health course for graduate students in the FNP program at NYU. She has lectured extensively throughout the greater NJ/NY area on women's health issues. 

Prior to joining faculty at NYU Meyers, Jeffers ran her own independent practice, Well Woman Care, LLC, serving the needs of women along the lifespan in her New Jersey community.  She also taught in the FNP program at Duke University.

Jeffers earned her DNP and BSN at Duke University, her MSN at Monmouth University, and her BS in kinesiology at the University of Maryland. Her doctoral research focused on a quality improvement project for midlife women experiencing menopausal symptoms. 


DNP - Duke University (2014)
MSN - Monmouth University (1998)
BS, Kinesiology - University of Maryland (1988)
BSN - Duke University (1982)


Faculty practice
Women's health
Primary care
Maternal global health

Professional membership

North American Menopause Society
Nurse Practitioners in Women's Health
Sigma Thea Tau International - Upsilon Chapter
Women's Health Advisory Council and Expert Panel

Honors and awards

Faculty Honors Awards

DNP Student speaker award, Duke University (2014)



Taylor, D., Woods, N. F., & Jeffers, L. S. (2017). Menopause. In J. Fitzpatrick (Ed.), Encyclopedia of nursing research (4th ed.). New York, NY: Springer Publishing Company. Retrieved from http://proxy.library.nyu.edu/login?url=https://search.credoreference.com/content/entry/spennurres/menopause/0?institutionId=577

Jeffers, L. (2017). In , & , Encyclopedia of nursing research (4th ed.).

Vagal and cardiac reactivity to psychological stressors in trained and untrained men

Spalding, T. W., Jeffers, L. S., Porges, S. W., & Hatfield, B. D. (2000). Medicine and Science in Sports and Exercise, 32(3), 581-591. 10.1097/00005768-200003000-00006
Purpose: The aim of this study was to determine whether higher aerobic fitness is associated with enhanced vagal influences on the myocardium, resulting in moderation of chronotropic cardiac activity during psychological stress and recovery. Method: Heart period (HP) and respiratory sinus arrhythmia (RSA) were obtained from 10 aerobically trained (AT) and 10 untrained (UT) college-aged men at rest and during three contiguous psychological challenges and 3 min of recovery. Ratings of perceived stress were obtained at the end of the rest period, at the midpoint of each stressor, and at 30 s into recovery. Time series methods were used to quantify RSA from the beat-to-beat HP series. Responsivity was assessed both in terms of absolute levels of activity and phasic changes in activity (task or recovery minus baseline). Results: Both groups reported similar levels of subjective stress throughout the experiment. The AT exhibited longer HP at rest and during psychological stress and recovery than did the UT. However, the groups did not differ on RSA at rest or during psychological stress and recovery, nor did they differ on phasic changes in RSA or HP during stress or recovery. Additionally, aerobic capacity was not correlated with absolute levels or phasic changes in RSA during psychological challenge for either group and, except in Min 2 for the UT, similar results were obtained for recovery. Conclusions: The results supported the hypothesis that, among young men, higher aerobic fitness is associated with longer HP at rest and during psychological stress and recovery. However, the lower cardiac chronotropic activation observed among the AT relative to the UT was not paralleled by a group difference in the amplitude of RSA. These results suggest that the group difference in HP was not mediated directly by the Vagal mechanisms manifested in the amplitude of RSA.